Fungus and antifungals

Vivian Imbriotis | June 13, 2026

Fungi include

  • Molds, which form hyphae (Aspergillus, mucor)
  • Yeasts, which remain unicellular (Candida albicans, other candida species, cryptococcus)
  • Dimorphs, which change between yeast and fungus forms (histoplasma)


Fungal cells have a

  • Cell wall, composed of \(\beta\)-glucans (polysaccarides) like plants
  • Cell membrane, stabilized by ergosterol (rather than cholesterol)

Ergosterol and glucan represent the two pathways to selective toxicity.

Azoles (Fluconazole, posiconazole)

  • Inhibit ergosterol synthesis \(\to\) loss of cell membrane integrity \(\to\) lysis
  • Hepatotoxic
  • Fluconazole \(\to\) yeasts (C Albicans and Cryptococcus)
  • Posiconazole \(\to\) C albicans, other candida sp,, crytococcus, aspergillus, mucor

Polyenes (Amphotericin)

  • Bind to ergosterol and open pores \(\to\) fluid influx \(\to\) lysis
  • Active against everything except some candida species
  • Hepto- and nephrotoxic, causes proximal renal tubular acidosis

Echinocadins (Anidulofungin)

  • Prevent \(\beta\)-glucan synthesis \(\to\) weaken cell wall \(\to\) lysis
  • Active against C albicans and other candida sp.
  • Anidulofungin removed by organ-independent spontaneous degradation
  • Minimal toxicity